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The ingredients, routines, lifestyle habits, and honest conversations that will change the way you relate to your skin, starting today.
In Part 1, we talked about the moment of realisation. The bathroom mirror moment. The one where your skin felt like it had quietly rewritten its own rules overnight, and you were the last to know.
If you missed it, go back and read it first, because this piece builds on that foundation.
Here, we move from the why to the what now.
And I want to start by saying something important: this is not a piece about ‘fixing’ your skin.
I have been there - standing in a chemist (drugstore) aisle, scanning labels for something that would turn back the clock, feeling vaguely panicked that my skin was ageing faster than I thought it should.
Buying more and more products, using actives that were too strong at the time, and wondering why my skin felt worse, not better.
What I have learned over the last decade or so, from my own skin, from formulating, from reading the research, and from listening to the 200+ women we surveyed about their real experiences, it is that the shift we all need is not about fighting harder.
It is about understanding better. And then using that understanding to support and nourish, not to ‘fight and fix’.
When you understand what is actually happening beneath the surface the guessing stops.
You stop chasing trends and start building something that actually works for your life, your preferences, the time you have and your budget.
That is what this piece is for.
Before we talk about products or routines, I want you to understand what you’re working with.
Because here is the thing nobody told us: midlife skin is not just older skin. It is a different skin.
Yours - but biologically, structurally, behaviourally different.
When estrogen signalling drops, and for most of us this begins in our late 30s with perimenopause, long before menopause is officially confirmed, multiple systems in the skin shift simultaneously.
While these shifts are completely normal, it’s this convergence that creates the confusing picture many of us see in the mirror.
"My skin felt like it was betraying me. I was doing everything right and it was getting worse." — one of the women we surveyed, age 47
She was not doing anything wrong. Her skin was responding to a changing biological environment. And that distinction changes everything about how we respond.
Estrogen supports the production of ceramides and fatty acids that form the skin's barrier - the protective film that keeps moisture in and irritants out. As estrogen declines, lipid production slows. The result is a compromised barrier that loses water more easily and reacts to things it once tolerated without issue.
What you feel: tightness after cleansing, sensitivity to products that were previously fine, skin that feels like it drinks moisturiser but stays dry. Skin that gets dehydrated more easily. Skin that takes longer to recover from breakouts or sensitivities and leaves more scarring.
In our survey, 68.5% of women reported persistent dehydration as one of their top concerns.
Collagen decline in early post-menopause is one of the most significant and well-documented shifts.
Some analyses cite reductions of up to approximately 30% within the first five years after menopause. The internal scaffolding thins. Lines that once faded overnight linger. Skin that once had tension and bounce begins to feel softer, not always in a way we welcome.
What you feel: lines that are deeper than you remember, skin that doesn't quite spring back the way it did, a softening around the jaw and under the eyes that feels unfamiliar.
Estrogen has antioxidant protective properties. As levels decline, the skin's natural defence against free radical damage - from UV, pollution, stress, inflammation - diminishes. The result can be accelerated oxidative damage to collagen fibres and cell DNA.
What you feel: dullness that feels deeper than just tiredness. In our survey, 80.5% of women named dull, tired-looking skin as a primary concern. This is not imagination. It is oxidative stress making itself visible.
Estrogen modulates inflammatory pathways. With declining levels, baseline inflammation increases and the skin's ability to self-regulate and recover slows. Small stressors - a new product, a stressful week, a broken night’s sleep - can cause bigger reactions than they used to.
What you feel: skin that flares unpredictably. Products that sting where they didn't before. A redness or reactivity that seems to have appeared from nowhere.
One of the most disorienting parts of midlife skin: acne. The same decade that brings dryness and collagen loss can also bring cystic spots - usually along the jaw and chin driven by the relative dominance of androgens as estrogen declines.
What you feel: standing at the bathroom mirror with a rich moisturiser in one hand and a spot treatment in the other, wondering how both could possibly be necessary at the same time. (I have stood in exactly that spot. It is maddening.) And they stay for weeks and weeks…
Hyaluronic acid production in the dermis is influenced by estrogen. As levels drop, the skin's water-binding capacity reduces. This is separate from the lipid barrier issue - you can address both, but you need to understand they are different problems requiring different solutions.
What you feel: skin that looks fine in morning light but dull and flat by afternoon. Makeup that settles into texture. A general flatness that hydrating serums help temporarily, but don't fully resolve.
Cell turnover slows. Dead cells accumulate on the surface. Melanocytes, the cells that produce pigment, become more erratic without estrogen's regulatory influence. Years-old sun damage that was sitting quietly beneath thicker collagen now surfaces and becomes more visible.
What you feel: a rougher surface texture, uneven tone, patches of pigmentation that seem more stubborn and persistent than before.
Understanding these seven shifts turns skincare from a guessing game into a conversation between you and your skin - and your priorities will depend on where you’re up to on your own journey.
There is something worth saying before we go any further, because I think it genuinely changes how you approach everything else in this piece.
Some of what we see in the mirror - the softening jawline, the deeper nasolabial folds, the hollowing under the eyes, the sense that our face has quietly changed shape - is not primarily a skincare problem. It is a structural one.
And no cleanser, serum or active ingredient, however well-formulated, can slow it down or reverse it.
Beneath the skin, three things are changing simultaneously: facial bone density is reducing, the fat pads that give the face its youthful volume are descending and thinning, and muscle tone is gradually shifting. These changes happen to all of us.
Estrogen decline accelerates them. And they are responsible for much of what we experience as the face "ageing fast" - even when our skin texture and tone are actually in good shape.
I say this not to be discouraging - quite the opposite. I say it because understanding the difference between what is structural and what is surface-level is genuinely freeing.
It stops you spending money and emotional energy chasing results that skincare was never designed to deliver. And it lets you use your routine for what it actually excels at: strengthening and supporting the barrier, reducing inflammation, and preserving the quality and resilience of what you have while improving the overall appearance of your skin.
What we can do at the surface level matters enormously. But it matters most when we are clear-eyed about what it is doing.
If you want to understand the full picture of what is happening beneath your skin - the bone, fat and muscle shifts and what, if anything, you can do about them - I've written about this in depth here. It's one of the most important pieces on the blog, and one of the most honest conversations I know how to have about ageing.
The skincare industry will happily sell you seventeen products and 10-step routines.
What I want to do is the opposite: help you understand which ingredients are doing the real structural work for estrogen-depleted skin, so you can build a focused, intentional routine rather than an expensive shelf of overlapping products.
I use a framework I call CHAMP to organise a complete routine, and ACE to identify the actives that make the biggest difference.
This is exactly how I take care of my own skin as well.
Let me walk you through both - not as a shopping list, but as a logic way for thinking about your skin.
For decades, many of us were conditioned to want that squeaky-clean feeling after washing our face.
If your skin feels tight after cleansing, that is not clean. It’s more of an indication that it’s been stripped or dried out. And for a barrier that is already compromised by declining lipid production, stripping it nightly is not helpful.
For estrogen-depleted skin, the cleanser is arguably the most important step - because getting it wrong undermines everything that follows. You want something that dissolves makeup and pollution without disturbing the skin's acid mantle and your barrier.
Oil-based cleansers and balms are particularly effective for this skin type because, like dissolves like, they lift sebum and impurities while simultaneously depositing lipids. The result is skin that feels comfortable, not tight, immediately after washing.
I created Radiance Reveal Cleansing Balm specifically because I wanted an oil-based cleanser that would not leave my changing skin feeling tight and reactive.
It uses 13 plant oils chosen for their affinity with mature, lipid-depleted skin
Dehydration in midlife skin is a two-layer problem. The first layer is surface dehydration - a lack of water in the uppermost layers of the epidermis. The second is deeper structural dehydration - reduced hyaluronic acid and glycosaminoglycan content in the dermis.
Addressing both requires layering. Start with a water-based serum or essence containing humectants - hyaluronic acid, sodium PCA, glycerin - applied to slightly damp skin to maximise absorption. Then follow with a product that seals the moisture in, rather than letting it evaporate - this is your moisturiser step.
This is where the structural work happens. For estrogen-depleted skin, three categories of actives are doing the heavy lifting.
We will cover these in detail in the next section below.
Many of us spent our 20s and 30s avoiding heavy creams - too greasy, too pore-clogging. That changes as you go into 40s and beyond.
With fewer lipids being produced naturally, a cream that delivers ceramides, fatty acids, and barrier-supportive ingredients is not a luxury; it is maintenance. For me it’s the bare minimum these days.
The question to ask of your current moisturiser: is it replacing what your skin is no longer making as efficiently?
Or is it just sitting on top without genuinely reinforcing the barrier?
We want BOTH the seal and the replenishment.
I know you’ve probably heard this hundreds of times by now… but it still stands as true as ever. UV exposure is the single largest accelerant of the visible changes estrogen decline makes possible.
As our collagen scaffolding thins, the visible legacy of decades of sun exposure emerges faster. SPF 30+ (for me it’s 50+) every morning - regardless of whether you plan to be outdoors - is not optional for skin in hormonal transition.
This does not need to be complicated.
A lightweight SPF that you will actually wear consistently is better than a superior formula you avoid because it feels heavy.
My absolute favourite is the Age Active Fluid from Heliocare.
When I look at the ingredients landscape for estrogen-depleted skin, three categories stand out as consistently evidenced, genuinely useful, and worth understanding in depth.
I think of them as the ACE actives:
= Vitamin A / Peptides as an alternative
= Vitamin C / Antioxidants
= Chemical Exfoliation / Acids
Retinoids (the Vitamin A family) are among the most researched actives in all of skincare.
The mechanism is well-established: they bind to retinoic acid receptors in skin cells, upregulating collagen synthesis, increasing cell turnover, and stimulating the production of glycosaminoglycans like hyaluronic acid.
For midlife skin specifically, this is profoundly relevant. Where estrogen once supported collagen production, topical Vitamin A can help maintain the stimulus for that same process. It is not a replacement for estrogen - nothing topical is - but it is the most evidence-backed tool we have for supporting the dermis from the outside.
A note on tolerance: midlife skin, with its compromised barrier and increased sensitivity, often cannot tolerate the retinoids that work well on younger, more resilient skin. Start with the lowest effective concentration.
Use it two to three nights a week at first, always on thoroughly dry skin. If you experience significant peeling or sensitivity, reduce frequency before reducing concentration.
As an alternative, or in addition to Retinoids, peptides are a gentler but genuinely useful complement. Signal peptides communicate with fibroblasts - the collagen-producing cells in the dermis - prompting them to produce more structural proteins. They work differently from retinoids and can be layered alongside them or used as an alternative for those whose skin cannot tolerate Vitamin A.
*** Peptides in skincare are a very big topic that we will cover separately in a dedicated blog.
If collagen is the scaffolding, antioxidants are the security system.
Every day, UV radiation, pollution, and internal stressors generate free radicals and inflammatory responses - unstable molecules that attack collagen fibres and accelerate the visible signs of ageing.
Vitamin C does two things for estrogen-depleted skin: it neutralises free radicals before they can damage existing collagen, and it supports collagen synthesis by acting as a cofactor in the process.
Applied every morning, it is essentially a shield for the structural work your evening actives are doing.
Vitamin C is also one of the most effective topical agents we have for addressing pigmentation - interrupting the enzyme (tyrosinase) that drives melanin production, and gradually brightening existing dark spots.
A few practicalities: Vitamin C is notoriously unstable. L-ascorbic acid, the most researched form, works best at a low pH (around 3.5), which can be irritating for sensitive midlife skin. Stabilised derivatives - ascorbyl glucoside, sodium ascorbyl phosphate - convert to active Vitamin C in the skin and are significantly gentler. Look for these if pure ascorbic acid stings.
*** We have a 3-part series on Vitamin C on the Beauty by Ané blog where you can learn more.
Slower cell turnover is one of the most visibly impactful changes in midlife skin - contributing to dullness, rough texture, and the persistence of pigmentation. Gentle, consistent exfoliation is the practical answer and one of the steps in your routine.
For estrogen-depleted skin, chemical exfoliation using AHAs and PHAs is generally preferable to physical scrubbing. Alpha-hydroxy acids (glycolic, lactic, mandelic) dissolve the bonds between dead cells and help them shed naturally. PHAs (gluconolactone, lactobionic acid) do the same with a larger molecular structure that penetrates more slowly - meaning they are significantly gentler on reactive or sensitive skin.
The goal is not aggressive peeling. It is gentle, consistent renewal - used two to three evenings a week - that keeps the surface clear, supports product absorption, and gradually improves texture and tone.
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* A NOTE ON PRODUCT AND INGREDIENT OVERLOAD One of the most common things I hear from women is that they have tried everything - and their skin has never been more reactive. Often, the problem is not what they are using, but how many things they are using simultaneously. Estrogen-depleted skin has a compromised barrier and a lower tolerance threshold. Introducing multiple actives at once - or layering acids with retinoids without giving the skin time to adapt - can cause chronic low-grade inflammation that counteracts the benefits of the ingredients themselves. My strong recommendation: introduce one active at a time, slowly. Give it four to six weeks. Then assess. Your skin will tell you what is working - if you give it the chance to adapt. |
One of the things that struck me most in our survey was how differently women ranked their concerns. Some were most bothered by firmness and structure. Others were primarily dealing with persistent dehydration and fine lines and wrinkles forming.
For others, it was acne - deeply ironic given that they were simultaneously struggling with dryness.
But for most it was that they had several concerns at the same time.
There is no universal routine for midlife skin.
There is a universal framework - CHAMP - but the specific products and actives you prioritise within it should reflect what your skin is actually telling you, what you feel you need and what your priorities are.
Morning: oil-based cleanse → Vitamin C serum → hydrating serum on damp skin → barrier-supportive moisturiser → SPF.
Evening: oil-based cleanse → hyaluronic acid serum on damp skin → seal with a lipid-rich moisturiser or face oil.
2–3 times per week, evenings: swap the serum step for a gentle chemical exfoliant to gently lift dead skin and improve luminosity.
The key principle: hydrate in layers, and always seal with your moisturiser and/or face oil.
Applying humectants without an occlusive layer on top allows water to evaporate from the skin surface, which can actually worsen dehydration.
Morning: gentle cleanse → Vitamin C serum → serum/moisturiser with peptides, ceramides and fatty acids → SPF.
Evening: gentle cleanse → retinoid (2–3 nights per week, on dry skin, low concentration to start) → barrier moisturiser. (peptides as an alternative to Retinoids)
On non-retinoid evenings: peptide serum → nourishing moisturiser and/or face oil.
The key principle: consistency over intensity.
Low-concentration retinoids used consistently over months deliver better results with fewer barrier disruptions than high-strength formulas used irregularly.
Morning: balm cleanse → antioxidant-rich serum (Vitamin E, niacinamide, or a stabilised Vitamin C) → ceramide-rich moisturiser → mineral SPF.
Evening: balm cleanse → a single barrier-repair moisturiser with ceramides and fatty acids. Nothing else.
Once the barrier has stabilised (this typically takes four to six weeks of this stripped-back routine): begin introducing a single PHA exfoliant, one night per week.
The key principle: before the skin can benefit from actives, it needs a functioning barrier. Reactive skin is a sign that the barrier needs support, not stimulation. Slow down before you layer up.
Morning: gentle oil-based cleanse → niacinamide serum (regulates sebum, reduces redness) → lightweight moisturiser → SPF.
Evening: oil-based cleanse (counterintuitive but important — it clears impurities without stripping) → salicylic acid or mandelic acid on affected areas, 2–3 nights per week → moisturiser.
Harsh, drying treatments make it worse. Anti-inflammatory, barrier-supportive care makes it better.
I want to be careful here, because this is the section that can tip from genuinely useful into overwhelming.
I have read plenty of wellness content that basically suggests your skin will only improve if you sleep nine hours, never experience stress, exercise every day, eat a perfectly varied diet, and drink three litres of water - all while balancing your nervous system, your blood sugar and cortisol levels in perfect harmony.
I am a 50-year-old entrepreneur running a business, writing a book, and managing the daily reality of life and a household.
We don’t have small children or teenagers in the house anymore and I’m finding it challenging - so anyone who does everything they have alongside actively taking care of children day in an day out have my biggest respect.
I do not always sleep nine hours (more like 5-6 most days). Stress is up and down; no day is perfect. And I have found that guilt about not living ‘perfectly’ because of all the things I’m not doing is itself a cortisol driver - which is literally counterproductive for your skin.
So let me give you the version of lifestyle-as-skincare that I actually believe in: small, consistent adjustments that support your skin without adding more to an already full plate.
If there is one thing I would say above everything else in this section - even above sleep, above stress management, above all of it - it is this: stay on top of inflammation as much as you possibly can.
We are not 'anti-ageing'.
Ageing is normal. It is physiological. It is not something to fight.
But we are absolutely anti-inflammation. Because chronic, low-grade inflammation - what researchers now call 'inflammaging' - is one of the single biggest drivers of accelerated skin ageing.
Here is why that matters so much right now: estrogen has genuine anti-inflammatory properties. It modulates the pathways that keep inflammation in check and helps the skin regulate and recover when it is triggered. As estrogen declines, that protection reduces. The threshold lowers.
Things that your skin once shrugged off - a stressful week, a broken night, a product it didn't love - now trigger a bigger response, and that response takes longer to resolve.
And here is the part that often gets missed: it is not just skincare products that trigger inflammation.
Poor sleep triggers it. Chronic stress triggers it. A diet high in ultra-processed foods triggers it. These are not separate lifestyle conversations - they are directly connected to what is happening in your skin at a deeper level - and how it ages.
So when we talk about sleep, movement, nutrition and stress in the sections below, I want you to hold this as the thread running through all of it: every one of these is, at its core, an inflammation management strategy.
Not because we are trying to be perfect. But because reducing the inflammatory load on skin that is already less protected than it used to be is one of the most meaningful things we can do for it - from the inside out.
Your skin does its most significant repair work during sleep - particularly in the first few hours, when growth hormone release peaks. This is when cells regenerate, collagen synthesis accelerates, and the barrier rebuilds.
For many women in perimenopause and menopause, sleep is exactly what is being disrupted - night sweats, insomnia, and shifts in circadian rhythm are among the most commonly reported symptoms. The knock-on effect on skin is real: poor sleep elevates cortisol, impairs barrier function, slows recovery, and increases inflammatory markers.
If sleep disruption is a feature of your life right now, treating it as a skincare priority - not a wellness add-on - reframes its importance.
A cooler sleeping environment, a consistent sleep window, and reducing evening screen exposure are all supported by evidence. And if sleep disruption is severe, this is worth raising with your GP - both for your skin and your overall health.
Elevated cortisol suppresses collagen synthesis, increases inflammation, and impairs barrier function. This is not metaphorical. It is a direct hormonal pathway from chronic stress to accelerated visible ageing.
Stress tolerance in midlife often reduces - partly because estrogen influences neurotransmitter systems that regulate emotional regulation, and partly because the cumulative weight of midlife tends to be objectively heavier. Acknowledging this is not weakness; it’s just helpful to understand the connection.
Five minutes of genuine quiet - breathing practice, a short walk, anything that you find helpful and fit into your schedule - has a measurable effect on cortisol patterns.
Exercise increases circulation, which delivers oxygen and nutrients to skin cells. It supports lymphatic drainage, which clears waste products. And it reduces systemic inflammation - one of the most meaningful drivers of accelerated skin ageing.
The barrier is low: a 10-minute brisk walk creates measurable cardiovascular response.
You do not need a structured workout. You need movement that raises your heart rate, consistently across the week.
A note on high-intensity training: HIIT and very intense exercise can temporarily spike cortisol. For some women in hormonal transition, very high exercise intensity can worsen inflammatory skin conditions.
If you notice breakouts or sensitivity after intense training, it may be worth adjusting intensity rather than eliminating movement altogether.
I am not a nutritionist, and I am wary of turning skincare into another domain where women are told their choices are inadequate.
But a few things are well-evidenced and simple:
I want to address this directly, because it is the elephant in the room of almost every conversation I have about midlife skin.
When we surveyed our community, a significant number of women said they felt pressured by the visible signs of ageing - and simultaneously confused about where to draw the line between skincare and professional/medical intervention.
Some felt that choosing injectables meant they had given up on their skin. Others felt that they may have left it too late.
Others felt that not having them meant they were not taking their appearance seriously enough.
Both of those framings are worth examining.
The question is not whether professional treatments are right or wrong.
The question is whether they are right for you - and whether you are making that choice from a place of informed agency.
Professional treatments - whether aesthetic procedures like laser or microneedling, or injectable treatments like botulinum toxin or filler - work on tissue that your daily skincare cannot reach or cannot address within a realistic timeframe.
Laser and radiofrequency treatments can stimulate deeper collagen production.
Microneedling creates controlled micro-injury that triggers repair responses. These are legitimate tools, and when performed well by qualified practitioners, the results are meaningful.
Injectable treatments address structure in ways topicals cannot: botulinum toxin relaxes muscles that pull skin into expression lines; hyaluronic acid filler restores volume that has been lost as fat pads thin and descend. In the right hands, the results look like well-rested, healthy skin — not frozen or inflated.
However - and this matters - no professional treatment works optimally on skin that is not well-maintained at the baseline. That’s why you will almost always be sold an at-home routine and products by your dermatologist/aesthetician.
A strong home skincare routine is not just complementary to professional treatments; it is the foundation that makes those treatments more effective and longer-lasting.
If you are considering professional treatments, here is what I would encourage you to think about:
I want to end this piece where I think it actually begins: in the emotional territory.
Because everything I have shared about CHAMP and ACE and barrier function and collagen is useful. But it will not stick - and it will not help - if the underlying relationship with your skin is one of frustration, blame, and urgency.
I know because I have been there. For a period in my early 40s, I was in what I can only describe as fix-and-fight mode.
Every new little line felt like a disappointment. Every new bit of pigmentation felt like evidence that I was not doing enough, or doing the wrong things, or had left it too late. I was spending time and money and emotional energy on skincare from a place of anxiety - and my skin wasn’t actually getting much better.
Stress elevates cortisol. Cortisol suppresses collagen synthesis. Anxiety about your skin is - physiologically, literally - making your skin age faster.
There is an irony here that I find both humbling and freeing: the most powerful thing you can do for your skin in midlife is to stop being at war with it.
That does not mean indifference. It does not mean giving up on skincare or accepting every change without response. It means shifting from a reactive, fear-driven approach to a curious, intentional and supportive one.
At some point - I cannot name the exact moment, but it was somewhere around 48 - stopped looking in the mirror trying to find what was wrong and started looking at what I actually saw.
I saw a face that has spent almost 50 years in the world. Skin that has carried me through enormous change - professionally, personally, geographically.
I saw the physical evidence of a life lived at some intensity.
I still care about my skin. I still use actives and SPF and a carefully formulated cleansing balm plus everything else we’re making to build our own CHAMP system.
I still read the research and think carefully about what I put on my face. But the feeling of it changed. More understanding. More considered. More interested.
That shift - from fighting to supporting, from fix to nourishing - is, I believe, the most important shift in the whole conversation about midlife skin.
What does it actually look like to support your skin through estrogen depletion, rather than fight it?
It looks like a cleanser that works with your barrier instead of stripping it. A moisturiser rich enough to replace what is no longer being produced efficiently. Actives that stimulate collagen production.
It looks like sleep is treated as a biological necessity rather than a luxury. Movement that brings oxygen and circulation without punishing your joints. Stress management and deeper self-care as a structural skincare step, not a self-indulgence.
It looks like an honest conversation with yourself about what you actually want - not what you think you should want, not what a filtered Instagram account suggests you should want, but what a genuine, informed decision and your vision for your future looks like for you.
Everything I have shared in this piece is topical and lifestyle-based. And there is an enormous amount you can do at that level.
But for many women reading this, there is a third leg to this conversation - one that has become more openly discussed in the last few years, and that sits at the intersection of hormonal health, skin biology, and deeply personal decision-making.
Hormone Replacement Therapy.
In our next piece - Part 3 of the Estrogen-Depleted Skin Series - we are going to look directly at the question many of you have raised: what does replacing lost estrogen actually do to the skin?
Is there meaningful evidence? What do the studies actually show?
And how do you think about it if you are undecided?
We will also look at the alternatives - phytoestrogens, targeted supplementation, and other approaches - for those for whom HRT is not suitable or not desired.
Understand why your skin suddenly feels different during perimenopause and menopause.
Learn how estrogen depletion and decline affects collagen, hydration, barrier function, and inflammation - and why this phase deserves its own conversation beyond 'normal ageing.'
Part 1 of the Estrogen-Depleted Skin Series.
Skincare marketing buzzwords like clean, natural, chemical-free, and non-toxic may sound reassuring - but what do they actually mean?
In this guide, we break down six common skincare marketing claims, explain why they’re misleading, and show you how to make informed choices based on cosmetic chemistry, not fear.
You’ve chosen your perfect Vitamin C - now, how do you use it effectively?
In Part 3 of our 'Vitamin C For Midlife Skin Decoded' guide I’ve compiled the 16 most common questions about Vitamin C in skincare, from layering with Retinol and Niacinamide and how long it can take to see results, to when is best to use it in your routine - and much more.
This Q&A is your cheat sheet for making Vitamin C work hardest for your midlife skin.
